350.4 ð ç« æ«éèš Summary
350.4.1 ð äžå¥è©±çžœçµ
IBS is most common functional GI disorder (10-15% prevalence globally, F > M 2:1)ïŒRome IV criteria: recurrent abdominal pain ⥠1 day/week à 3 months + 2+ of (related to defecation, change in stool frequency, change in stool form); symptom onset ⥠6 monthsïŒsubtypes by Bristol Stool Scale: IBS-D (diarrhea, > 25% loose), IBS-C (constipation, > 25% hard), IBS-M (mixed both ⥠25%), IBS-U (unspecified)ïŒpathophysiology multifactorialïŒvisceral hypersensitivity + altered motility + altered microbiome (dysbiosis) + low-grade inflammation + brain-gut axis dysfunction + psychosocial stress + post-infectious IBS + genetic predispositionïŒdiagnosisïŒclinical + Rome IV + exclude alarm features (⥠50 yo, weight loss, hematochezia, anemia, family hx GI cancer, nocturnal, persistent severe); fecal calprotectin (distinguishes IBD from IBS) + celiac serology (especially IBS-D) + TSHïŒtreatment foundationïŒeducation + reassurance + low FODMAP diet (most evidence) + lifestyle (stress, exercise)ïŒIBS-C medicationïŒPEG + linaclotide (Linzess GC-C) + lubiprostone + plecanatide + tenapanor (Ibsrela NHE3 FDA 2019) + prucalopride (5-HT4)ïŒIBS-D medicationïŒloperamide + rifaximin (Xifaxan 14-d course) + eluxadoline (avoid pancreatitis/cholecystectomy hx) + alosetron (women, REMS) + bile acid sequestrants for bile acid diarrheaïŒpain managementïŒTCAs low-dose for IBS-D (amitriptyline 10-25 HS), SSRIs for IBS-C, antispasmodics (dicyclomine, peppermint oil), pregabalin/gabapentinïŒpsychological therapiesïŒCBT (most evidence) + gut-directed hypnotherapy + mindfulnessïŒother functional GIïŒfunctional dyspepsia (Ch348), functional constipation (slow transit / outlet dysfunction â biofeedback / normal transit), functional diarrhea, cyclic vomiting syndrome (migraine association, amitriptyline preventive), cannabinoid hyperemesis syndrome (heavy cannabis + hot shower relief â cessation cure), globus sensation, functional heartburn (Rome IV); 2024 emerging: microbiome modulation + FMT trials + gut-brain axis modulatorsã
350.4.2 ð æ²»ç粟èŠ
- dietïŒlow FODMAP diet (Fermentable Oligosaccharides Disaccharides Monosaccharides And Polyols) 3 phases (restriction 4-6 wk â reintroduction â personalization) â most evidence; dietitian guidance helpful
- IBS-C medicationsïŒpolyethylene glycol (PEG) first-line laxative, linaclotide (Linzess) guanylate cyclase C agonist, lubiprostone (chloride channel activator), plecanatide (GC-C), tenapanor (Ibsrela) NHE3 inhibitor FDA 2019, prucalopride (Motegrity) 5-HT4 agonist
- IBS-D medicationsïŒloperamide (mu-opioid receptor agonist symptomatic), rifaximin (Xifaxan) non-absorbed antibiotic 14-d course (TARGET-3 supports re-treatment), eluxadoline (mu-opioid + delta â avoid pancreatitis/cholecystectomy history), alosetron (5-HT3 antagonist women REMS program), bile acid sequestrants (cholestyramine, colesevelam) for bile acid diarrhea
- pain managementïŒTCAs (amitriptyline 10-25 mg HS) for IBS-D especially, SSRIs for IBS-C especially, antispasmodics (dicyclomine, hyoscyamine, peppermint oil), pregabalin/gabapentin off-label visceral pain
- psychologicalïŒCBT (cognitive behavioral therapy â most evidence) + gut-directed hypnotherapy + mindfulness + acceptance commitment therapy
- functional constipation outlet dysfunctionïŒbiofeedback is effective treatment
- cannabinoid hyperemesis syndromeïŒcessation curative + topical capsaicin + antiemetics + hot showers
- cyclic vomiting syndrome preventiveïŒamitriptyline, propranolol, topiramate (migraine-related)
350.4.3 ð¯ ç§é«åž«çèåæé
- IBS Rome IV criteriaïŒrecurrent abdominal pain ⥠1 day/week à 3 months + 2+ of (related to defecation, change in stool frequency, change in stool form) â onset ⥠6 months
- alarm features warranting workupïŒâ¥ 50 yo new onset, weight loss, hematochezia, anemia, family history GI cancer, nocturnal symptoms â colonoscopy + comprehensive workup
- fecal calprotectin is key biomarker: distinguishes IBD (elevated > 250) from IBS (normal) â first-line screening
- low FODMAP diet is most evidence-based dietary intervention: 3 phases (restriction 4-6 wk â reintroduction â personalization); 50-75% improve with dietitian guidance
- IBS-C medications by mechanismïŒlinaclotide + plecanatide (GC-C agonists), lubiprostone (ClC-2 activator), tenapanor (NHE3 inhibitor), prucalopride (5-HT4 agonist) â multiple options, individualize
- IBS-D medicationsïŒrifaximin (Xifaxan, 14-d course) â non-absorbed; can re-treat per TARGET-3; eluxadoline avoid pancreatitis/cholecystectomy history (Viberzi REMS); alosetron REMS program (women only, ischemic colitis risk)
- TCA (amitriptyline 10-25 HS) for IBS-D pain â peripheral anticholinergic + central modulation; SSRIs for IBS-C â accelerate gut transit
- CBT (cognitive behavioral therapy) has strongest evidence for psychological intervention in IBS; gut-directed hypnotherapy also effective; gut-brain axis modulation
- cannabinoid hyperemesis syndrome (CHS)ïŒheavy chronic cannabis + cyclic vomiting + relief from hot showers/baths (pathognomonic) + cessation curative; topical capsaicin + antiemetics; growing recognition with legalization
- functional constipation subtypesïŒslow transit + outlet dysfunction (pelvic floor dyssynergia â biofeedback effective) + normal transit â anorectal manometry + balloon expulsion + colonic transit study for evaluation