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- è: Mycobacterium leprae + M. lepromatosis (Mexico, Caribbean) â acid-fast bacilli, intracellular, cannot culture in vitro (grow only in 9-banded armadillo + mouse footpad)
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åž:
- WHO 2024: ~ 175,000 new cases/yr globally
- High burden: India (60%), Brazil (15%), Indonesia
- Taiwan: very few; mostly imported
- Transmission: prolonged close contact + droplet (less infectious than TB); ~ 95% humans innately resistant
- Incubation: long (3-10 years average)
- Reservoir (zoonotic component): armadillo (USA southern, Texas, Louisiana, Florida)
- Spectrum (Ridley-Jopling):
- Tuberculoid (TT): strong CMI, few well-defined hypopigmented anesthetic plaques, few bacilli, peripheral nerve thickening
- Borderline (BT, BB, BL): intermediate
- Lepromatous (LL): weak CMI, many bacilli, diffuse skin + multi-organ + leonine facies + saddle nose + glove-and-stocking neuropathy + ocular + nasal + bone changes
- Clinical features:
- Hypopigmented anesthetic skin plaques (loss of sensation key â distinguishes from other dermatoses)
- Peripheral nerve thickening (ulnar, posterior auricular great, common peroneal, posterior tibial, supraorbital)
- Neuropathic injury (Charcot foot, claw hand, lagophthalmos, foot drop)
- Diagnosis:
- Clinical (hypopigmented anesthetic patch + nerve thickening = suspect)
- Slit-skin smear AFB (positive lepromatous, negative tuberculoid)
- Skin biopsy histology + Fite stain
- PCR (specialized labs)
- Treatment (WHO MDT):
- PB (paucibacillary, TT/BT): Rifampin 600 mg monthly + Dapsone 100 mg daily à 6 mo
- MB (multibacillary, BB/BL/LL): Rifampin 600 mg monthly + Dapsone 100 mg daily + Clofazimine 300 mg monthly + 50 mg daily à 12 mo
- Single skin lesion PB: ROM regimen â Rifampin + Ofloxacin + Minocycline à 1 dose alt
- Reactions:
- Type 1 (Reversal Reaction): T-cell mediated; pre-existing lesion inflamed; treat with steroid
- Type 2 (ENL â Erythema Nodosum Leprosum): immune complex; new tender erythematous nodules + fever + neuritis; thalidomide + steroid
- Prevention:
- 2018 WHO PEP: single-dose rifampin 600 mg for household contacts (reduces incidence ~ 50%)
- BCG vaccine â some efficacy
- Active surveillance + early treatment
1ïžâ£ 现èåž
- M. leprae â acid-fast bacillus, intracellular (macrophage, Schwann cell)
- Cannot culture in vitro â only in armadillo or mouse footpad
- 30-50% genome decay (degenerate metabolism; obligate parasite)
- Optimal at 30-33°C (cooler body parts â skin, peripheral nerves, anterior chamber eye, nasal mucosa, ears)
- M. lepromatosis â Mexico + Caribbean variant, often diffuse lepromatous form
Pathogenesis
- Macrophage uptake â æ
¢ granuloma formation
- Schwann cell tropism (cooler body parts) â demyelination â nerve thickening + neuropathic injury
- Spectrum from cell-mediated (TT) to humoral (LL) immune response
2ïžâ£ Spectrum (Ridley-Jopling)
Tuberculoid (TT)
- Strong CMI, granulomatous response
- Few well-defined hypopigmented anesthetic plaques (1-3 lesions)
- Peripheral nerve thickening
- Few bacilli on slit-skin smear (paucibacillary)
- Less infectious
Borderline Tuberculoid (BT)
- More lesions
- Variable bacterial load
Borderline Borderline (BB)
Borderline Lepromatous (BL)
- Many lesions, increasing bacilli
Lepromatous (LL)
- Weak CMI, Th2 humoral response
- Many bacilli (multibacillary, high infectivity)
- Diffuse skin involvement, infiltration
- Leonine facies (loss of eyebrows, infiltration of forehead + cheeks)
- Saddle nose (nasal cartilage destruction)
- Madarosis (eyebrow loss)
- Glove-and-stocking neuropathy (symmetric)
- Hands + feet: clawing, ulceration, autoamputation
- Eye: lagophthalmos, corneal anesthesia, iritis, cataracts
- Nasal: chronic rhinitis, congestion, perforation
- Bone: osteo destruction
- Genital: orchitis (infertility in men)
Diffuse Lepromatous
- Variant â diffuse infiltration without discrete lesions
- M. lepromatosis often
- Lucioâs phenomenon (vasculitis necrosis)
3ïžâ£ Clinical Features
Skin
- Hypopigmented or erythematous plaques with loss of sensation (KEY distinguishing feature)
- Borders well-defined (TT) or ill-defined (LL)
- Anhidrosis (loss of sweating in lesion)
- Hair loss in lesion
- Distribution: cooler body parts (face, ears, extensor surfaces; usually spare axilla, groin, perineum)
Peripheral Nerve
- Nerve thickening (palpable):
- Ulnar at elbow
- Posterior auricular great in neck
- Common peroneal at fibular neck
- Posterior tibial at medial malleolus
- Supraorbital, infraorbital, mandibular in face
- Radial cutaneous at wrist
- Sural in lower leg
- Sensory deficits: glove-and-stocking (LL) or focal in lesion (TT)
- Motor deficits:
- Claw hand (ulnar + median palsy â MCP hyperextended, IP flexed)
- Foot drop (common peroneal)
- Lagophthalmos (CN VII facial â orbicularis oculi)
- Lateral popliteal: foot drop
- Charcot joint: neuropathic destruction
- Plantar ulcers: unfelt repetitive trauma
Eye
- Lagophthalmos + corneal anesthesia + dry eye â corneal damage
- Iritis, scleritis, uveitis (LL)
- Cataracts
- Blindness in advanced
Nasal + Upper Airway
- Chronic rhinitis
- Septum perforation
- Saddle nose (chronic LL)
Bone
- Cortical thinning, resorption
- Osteoperiostitis
Other
- Orchitis (LL, men) â infertility
- Lymphadenopathy
- Hepatomegaly (LL)
4ïžâ£ Diagnosis
Clinical Criteria (WHO)
- ⥠1 of:
- Hypopigmented or erythematous skin lesion with definite sensory loss
- Thickened peripheral nerve(s)
- Positive slit-skin smear (AFB)
Slit-Skin Smear
- AFB stain (acid-fast bacilli)
- Sites: earlobes, elbow, knee, suspected lesions
- Positive in LL/BL (multibacillary)
- Negative in TT/BT (paucibacillary)
Skin Biopsy
- Histology: granulomas (TT) vs foam cells (LL)
- Fite stain (modified AFB) for M. leprae
- âGlobiâ â clusters of bacilli (LL)
Other
- PCR (specialty labs)
- Phenolic glycolipid I (PGL-I) antibody (rare clinical)
- Nerve biopsy (rare)
- Imaging: nerve thickening on US
5ïžâ£ Treatment (WHO MDT)
PB (Paucibacillary): TT, BT, Single Lesion
- Rifampin 600 mg PO once monthly (supervised)
- Dapsone 100 mg PO daily (self-administered)
- Duration: 6 months
- Cure rate > 95%
MB (Multibacillary): BB, BL, LL
- Rifampin 600 mg PO once monthly (supervised)
- Dapsone 100 mg PO daily
- Clofazimine 300 mg PO once monthly + 50 mg PO daily
- Duration: 12 months (2018 WHO update â previously 24 mo)
Single Skin Lesion (ROM)
- Rifampin 600 mg + Ofloxacin 400 mg + Minocycline 100 mg
- Single dose (alt to 6-mo PB regimen)
- Logistic advantage for remote / single-lesion cases
Pediatric Dosing
- Weight-based
- Same drugs
- Rifampin caution if hepatic issues
Pregnancy + Lactation
- Standard MDT OK
- Dapsone safe; rifampin safe; clofazimine â neonatal skin discoloration (resolves)
G6PD Deficiency + Dapsone
- Dapsone causes hemolysis in G6PD def
- Screen G6PD before starting
- Substitute clofazimine + minocycline if G6PD positive
Resistance
- Emerging in some regions (~ 5% MB cases)
- Resistance testing â specialized labs
- Alternative drugs: ofloxacin, minocycline, clarithromycin
Free MDT
- WHO provides MDT free of charge globally through Novartis donation
- Available in all endemic countries
6ïžâ£ Leprosy Reactions
Type 1: Reversal Reaction
- Cell-mediated, sudden inflammation of pre-existing lesions
- Skin: erythema, swelling, ulceration of existing patches
- Nerve: acute neuritis with pain + functional loss
- Onset: during or after MDT, especially BT/BB/BL
- Treatment: prednisolone 40-60 mg PO qd taper à 12-24 weeks
- Continue MDT
- Surgical decompression if severe nerve
Type 2: Erythema Nodosum Leprosum (ENL)
- Immune complex deposition (LL/BL)
- New tender erythematous nodules + fever + arthralgia + neuritis + iritis + lymphadenitis
- Glomerulonephritis, orchitis, vasculitis
- Recurrent episodes
- Treatment:
- Mild: NSAID
- Moderate-severe: prednisolone + thalidomide (highly effective for ENL)
- Thalidomide 100-400 mg PO qd â pregnancy contraindicated (teratogenic â STEPS program in US)
- Clofazimine (anti-inflammatory) adjunct
- Continue MDT
Lucioâs Phenomenon
- M. lepromatosis diffuse LL
- Vasculitis with skin necrosis
- High mortality
- Treatment: MDT + steroid
7ïžâ£ Prevention + Public Health
2018 WHO Post-Exposure Prophylaxis (PEP)
- Single dose rifampin 600 mg PO for household + close contacts of new cases
- Reduces incidence in contacts ~ 50% (LPEP trial)
- Cost-effective
- Integrating in many endemic programs
BCG Vaccine
- Some efficacy against leprosy (~ 50% â variable)
- Routine in some endemic
- BCG revaccination â some evidence
Active Surveillance
- Contact screening (3-5 years post case)
- Self-skin checks in endemic
- Early diagnosis + treatment
Stigma Reduction
- Lifelong impact of disability + stigma
- Counseling, vocational rehabilitation
- Term âHansenâs diseaseâ (after Gerhard Hansen who identified M. leprae 1873) preferred over âleprosyâ in many contexts
WHO Roadmap
- Global leprosy strategy 2021-2030: end leprosy as public health problem
- Zero new pediatric cases with disability
- Zero new transmission