293.1.0.1.5.2 Primary Prevention (Pre-Treatment)
- Cardiac rehabilitation
- BP control, statin if indicated
- Stop smoking
- Weight management
- Exercise during treatment
Mechanism: oxidative stress, topoisomerase IIβ inhibition, mitochondrial damage
Dose-Dependent Cardiotoxicity: - Doxorubicin: > 250-350 mg/m² cumulative â â HF risk - Older threshold 550 mg/m² (less safe) - Liposomal form less cardiotoxic
Time Course: - Acute (< 1 week): rare, arrhythmia - Early-onset chronic (within 1 yr): more common - Late-onset (years later): childhood cancer survivors
Clinical: - Asymptomatic LV dysfunction â HF - Mostly HFrEF - Often irreversible (vs trastuzumab)
Risk Factors: - Cumulative dose - Age extremes - Pre-existing CV disease - Combination with trastuzumab, radiation - Concomitant cardiotoxic agents
Prevention: - Dexrazoxane: iron chelator, â cardiotoxicity (Class IIa for high-risk + > 300 mg/m²) - Cardiac risk assessment - Lower-dose regimens - Liposomal formulation - ARNI/ACEi + β-blocker + SGLT2i (primary prevention emerging)
Monitoring: - Echo + troponin baseline + during + after treatment - 6-12 months post-completion - Annual surveillance for high-risk
Mechanism: blocks HER2 â impairs cardiomyocyte repair
Cardiotoxicity: reversible LV dysfunction (NOT dose-cumulative) - Often after anthracycline â synergistic - Reversible in 50-70%
Monitoring: echo every 3 months during treatment
Treatment: hold trastuzumab, start HF therapy, re-challenge after recovery often successful
Toxicity: - HTN (10-30%) - Proteinuria - Arterial thromboembolism - Hemorrhage - LV dysfunction - Thrombotic microangiopathy
Management: BP control, monitor UA, watch for thromboembolism
VEGF TKIs (sunitinib, sorafenib, axitinib, pazopanib): - HTN - LV dysfunction (~ 10%) - QT prolongation - Arrhythmia
BCR-ABL TKIs (imatinib, dasatinib, nilotinib, ponatinib, bosutinib): - Ponatinib: arterial thrombosis, MI, stroke (~ 30%) - Nilotinib: PAD, vascular events - Dasatinib: pulmonary HTN (rare, may reverse) - Imatinib: relatively safer
BTK Inhibitors (ibrutinib, acalabrutinib): - AF (10-15%) - Bleeding (esp on anticoagulation) - HTN - Ventricular arrhythmias
Management: cardio-oncology consultation, optimize CV risk factors, switch drug if severe
Drugs: pembrolizumab, nivolumab, ipilimumab, atezolizumab, durvalumab, cemiplimab
Cardiotoxicity: - Myocarditis (rare 0.5-1.5%, but 50% mortality if severe) - Pericarditis, pericardial effusion - Arrhythmia - Conduction abnormalities (heart block) - Vasculitis (uncommon) - Takotsubo
Risk Factors: - Combination ICI (e.g., nivolumab + ipilimumab) - Pre-existing CV disease - DM - Multi-irAE patient
Clinical: - Onset typically 2-12 weeks after start - Symptoms: dyspnea, chest pain, palpitations, syncope, fatigue - Severe: cardiogenic shock, VT/VF, complete heart block
Diagnosis: - Troponin (sensitive) - ECG: arrhythmia, conduction - BNP/NT-proBNP - Echo: LV dysfunction - CMR: T2 hyperintensity + LGE (Lake Louise criteria) - EMB (endomyocardial biopsy): gold standard - NEW: 18F-FDG PET-CT for cardiac inflammation
Treatment: - Stop ICI - High-dose corticosteroids (methylprednisolone 1 g/d IV Ã 3 d, then 1 mg/kg/d taper) - Refractory: infliximab (anti-TNF), MMF, ATG, abatacept, JAK inhibitors (tofacitinib) - HF therapy + supportive - ECMO bridge in severe
Chest radiation (especially mantle-field in Hodgkinâs, breast): - Premature CAD: years later - Pericardial disease: acute + chronic (constrictive) - Valvular disease: usually AR / AS late - Conduction abnormalities: AV block - Cardiomyopathy: restrictive - Carotid stenosis + premature atherosclerosis
Risk: dose > 30 Gy + young at exposure
Surveillance: lifelong CV follow-up; echo + stress testing periodically; consider statin