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Definition
- Heart rate > 100 bpm + narrow QRS (< 120 ms typically)
- Originates above ventricles (atria + AV junction)
- Distinct from sinus tachycardia (physiologic)
Major SVT Types
Sinus Tachycardia
- Compensatory (fever, hypovolemia, anemia, pain, exercise, hyperthyroidism, drugs, anxiety, sepsis)
- Pathologic: inappropriate sinus tachycardia, POTS
- Gradual onset/offset
Atrial Fibrillation (AF)
- Most common sustained arrhythmia (Ch 264 dedicated)
- Irregularly irregular
- No P waves
Atrial Flutter (AFL)
- âSaw-toothâ pattern (esp II, III, aVF)
- Atrial rate ~ 300 bpm
- Often 2:1 conduction â ventricular rate 150
- Treatment: cavotricuspid isthmus ablation (typical, counterclockwise) very effective
Atrioventricular Nodal Reentrant Tachycardia (AVNRT)
- Most common paroxysmal SVT (~ 60%)
- Slow + fast pathway in AV node
- Re-entry within AV node
- Regular narrow complex; rate 150-220
- Retrograde P often hidden in QRS or just after (pseudo-S in II, III, aVF; pseudo-Râ in V1)
- Sudden onset / offset
Atrioventricular Reentrant Tachycardia (AVRT)
- Accessory pathway-mediated (WPW)
- Macro re-entry circuit (AV node + accessory pathway)
- Orthodromic (~95%): conducts down AV node + up accessory pathway â narrow QRS
- Antidromic (~5%): conducts down accessory pathway + up AV node â wide QRS (mimics VT)
- WPW on baseline ECG (short PR + delta wave + wide QRS) when not in SVT
Atrial Tachycardia (AT)
- Focal automaticity (atrial focus other than SAN)
- Variable P wave morphology (different from sinus P)
- Multiple foci: multifocal AT (⥠3 P morphologies â often COPD, electrolytes)
- May or may not respond to adenosine
- Sometimes warm-up phenomenon (gradual rate increase)
Junctional Tachycardia
- Originates in AV junction
- HR usually 60-130 (junctional rate)
- AV dissociation possible
- Often digoxin toxicity, post-cardiac surgery
Acute Management
Stable Patient
- Vagal maneuvers: Valsalva, modified Valsalva (REVERT trial â better), carotid sinus massage
- Adenosine 6 mg IV rapid push â 12 mg if needed â may give 18 mg
- Calcium channel blocker IV (verapamil, diltiazem) for AVNRT, AVRT (not WPW+AF)
- β-blocker IV alternative
- Cardioversion if unstable (synchronized)
Unstable Patient (Hypotension, Chest Pain, Pulmonary Edema, Altered Mental Status)
- Synchronized cardioversion (50-100 J biphasic)
Antidromic AVRT (Wide-Complex SVT)
- Treat as VT until proven otherwise
- Procainamide IV
- Avoid AV nodal blockers (worsens â accessory pathway becomes only route)
WPW + AF (Pre-Excited AF) â Special Caution
- Avoid AV nodal blockers (BB, CCB, digoxin, adenosine)
- Reason: blocks AV node â accessory pathway conducts unimpeded â very rapid ventricular response â may degenerate to VF
- Procainamide IV or cardioversion preferred
Chronic Management
Recurrent Symptomatic SVT
- Catheter ablation = curative for AVNRT, AVRT, AFL, AT, idiopathic SVT
- Success rate > 95% for AVNRT + typical AFL; > 90% AVRT
- Outpatient procedure (4-6 hr typically)
- Reduces / eliminates antiarrhythmic drug use
Drug Therapy (Alternative to Ablation)
- β-blockers (first-line for many)
- Calcium channel blockers (verapamil, diltiazem)
- Flecainide (for refractory; avoid in structural heart disease)
- Propafenone (similar)
- Amiodarone (last resort due side effects)
WPW Management
- Catheter ablation of accessory pathway = curative + standard for symptomatic
- Asymptomatic WPW: risk stratification (younger high-risk preference for ablation)
Specific Conditions
Multifocal Atrial Tachycardia (MAT)
- ⥠3 different P morphologies
- COPD, hypoxia, electrolyte abnormalities
- Treat underlying (oxygen, bronchodilators, K+, Mg2+)
- IV magnesium, β-blockers (caution in COPD), CCB
Inappropriate Sinus Tachycardia
- Resting HR > 90 bpm with no obvious cause
- Often young women
- POTS (Postural Orthostatic Tachycardia Syndrome) â orthostatic increase > 30 bpm
- Treatment: lifestyle, ivabradine, beta-blocker
1ïžâ£ ECG Approach to Narrow Complex Tachycardia
Step 1: Regular vs Irregular
- Irregular: AF, atrial flutter with variable block, MAT
- Regular: AVNRT, AVRT, atrial flutter (constant block), AT, junctional, sinus tachycardia
Step 2: P-Wave Analysis
Visible P Before Each QRS
- Sinus tachycardia (P normal morphology)
- Atrial tachycardia (P abnormal morphology)
- Atrial flutter with 1:1 conduction (rare)
No Visible P or P Hidden
- AVNRT typical (retrograde P hidden in QRS or just after)
- Junctional tachycardia
Inverted P (Retrograde)
- AVNRT, AVRT (retrograde conduction)
Step 3: Response to Adenosine
- Sinus tachycardia: gradual slowing
- AVNRT: terminates (often)
- AVRT: terminates (often)
- Atrial flutter / AT: unmasks flutter waves / no termination but slows ventricular response (transient)
- Sinus: gradual slowing
- MAT: no termination but slows
Step 4: Other Features
- Onset / offset (sudden vs gradual)
- Provocation factors
- Family history
- Baseline ECG (WPW pattern?)
2ïžâ£ AVNRT (AV Nodal Reentry Tachycardia)
Mechanism
- Slow pathway + fast pathway in AV node (dual AV nodal physiology)
- Typical AVNRT (90%): slow path antegrade + fast path retrograde â atrial activation simultaneous with ventricular
- Atypical AVNRT (10%): fast path antegrade + slow path retrograde
- Re-entry circuit within AV node
Clinical
- Sudden onset palpitations
- Often young + middle-age women (3:1 women:men)
- Rate 150-220
- Triggered by ectopic beats, stress, caffeine, exercise
- Self-limited or terminated with vagal maneuver / adenosine
ECG
- Regular narrow complex tachycardia
- No discrete P waves (often hidden in QRS or just after â pseudo-S in II, III, aVF or pseudo-Râ in V1)
- Rate 150-220
Acute Management
- Vagal maneuvers (Valsalva or modified Valsalva â REVERT trial; carotid sinus massage)
- Adenosine 6 mg IV rapid push â 12 mg if needed
- IV calcium channel blocker (verapamil, diltiazem) alternative
- IV β-blocker
- Cardioversion if unstable
Chronic / Recurrent
- Catheter ablation = first-line for symptomatic recurrent (success > 95%, complications < 1%)
- Targets slow pathway
Catheter Ablation
- Slow pathway modification (preferred â preserves AV node)
- Outpatient
- Major complications: AV block requiring pacemaker (1%)
3ïžâ£ AVRT (AV Reciprocating Tachycardia / WPW)
Mechanism
- Accessory pathway (Bundle of Kent) connecting atria + ventricles outside AV node
- Re-entry circuit using AV node + accessory pathway
Orthodromic AVRT (~ 95%)
- Antegrade conduction: AV node â ventricle â accessory pathway retrograde
- Narrow QRS during tachycardia
- P wave after QRS (retrograde)
Antidromic AVRT (~ 5%)
- Antegrade conduction: accessory pathway â ventricle â AV node retrograde
- Wide QRS during tachycardia (resembles VT)
- Treat as VT until proven otherwise
Wolff-Parkinson-White (WPW) Syndrome
- Pre-excitation pattern on baseline ECG:
- Short PR (< 120 ms)
- Delta wave (slurred upstroke of QRS)
- Wide QRS (> 110 ms)
- Conduction down accessory pathway preempts AV node
Acute AVRT Management
Orthodromic (Narrow QRS)
- Vagal maneuvers + adenosine + AV nodal blockers (similar to AVNRT)
Antidromic (Wide QRS)
- Treat as VT until proven otherwise
- Procainamide IV (slows accessory pathway)
- Cardioversion if unstable
Pre-Excited AF (WPW + AF)
- AVOID AV nodal blockers (BB, CCB, digoxin, adenosine)
- Rationale: blocking AV node leaves accessory pathway â very rapid ventricular response
- May degenerate to VF
- Procainamide IV or cardioversion preferred
Chronic Management
- Catheter ablation of accessory pathway = curative + standard for symptomatic
- Success > 95%
- Risk stratification for asymptomatic WPW:
- Younger, athletic, high-risk occupation â consider ablation
- EP study + risk assessment of accessory pathway refractoriness
Risk Stratification â Sudden Death Risk in WPW
- Short refractory period of accessory pathway (< 250 ms)
- Multiple accessory pathways
- Septal location
- Familial AVRT
- Symptomatic (palpitations, syncope, AF)
- Lowest risk: asymptomatic adults > 35 yr with no concerning features
4ïžâ£ Atrial Flutter (AFL)
Mechanism
- Macro-reentry circuit in atria
- Typical (counterclockwise): cavotricuspid isthmus circuit (right atrium); negative flutter waves in II, III, aVF + positive in V1
- Atypical: various circuits
ECG
- Saw-tooth pattern (most evident II, III, aVF)
- Atrial rate ~ 300 bpm
- Often 2:1 AV conduction â ventricular rate ~ 150 bpm
- 3:1 (rate 100) or 4:1 (rate 75) also seen
- Variable conduction â irregular ventricular response
Acute Management
- Vagal maneuvers + adenosine (slows AV conduction, unmasks flutter waves)
- IV β-blocker or CCB (rate control)
- Cardioversion if unstable
- Cardioversion + anticoagulation considerations (similar to AF)
Chronic Management
- Cavotricuspid isthmus ablation = highly effective for typical atrial flutter (> 95% success)
- Antiarrhythmic alternative
- Rate control (β-blocker, CCB) + anticoagulation (CHA2DS2-VASc)
Anticoagulation
- Same principles as AF
- CHA2DS2-VASc score for stroke risk
5ïžâ£ Atrial Tachycardia (AT)
Mechanism
- Focal automaticity (atrial focus other than SAN)
- May respond to / not respond to adenosine (depends on mechanism)
- Atrial rate typically 100-250
ECG
- P wave morphology different from sinus
- P-P regular usually
- Sometimes âwarm-up phenomenonâ (gradual rate increase)
Multifocal Atrial Tachycardia (MAT)
- ⥠3 different P morphologies
- COPD common, hypoxia, theophylline, hypokalemia, hypomagnesemia
- Irregular rhythm
- Treatment: oxygen, treat COPD, correct electrolytes, IV magnesium, β-blocker (caution in COPD)
Sustained Atrial Tachycardia
- Catheter ablation if symptomatic
- β-blocker / CCB / antiarrhythmics
6ïžâ£ Vagal Maneuvers + Adenosine
Vagal Maneuvers
Valsalva Maneuver
- Sustained expiratory effort against closed glottis
- ~ 25% effective alone
Modified Valsalva (REVERT Trial)
- Standard Valsalva + leg elevation immediately after
- More effective (43% vs 17% standard)
- Become first-line vagal maneuver
Carotid Sinus Massage
- Unilateral, 5-10 seconds
- Risk: stroke (elderly with carotid disease)
- Auscultate for bruit first
- Avoid in significant carotid disease
Adenosine
Dose
- 6 mg IV rapid push (followed by saline flush) â if no response in 2 min:
- 12 mg IV â if no response:
- 18 mg IV (some protocols)
Mechanism
- Brief AV node block
- Terminates re-entrant SVT (AVNRT, AVRT)
- Unmasks atrial activity in flutter / AT
Side Effects
- Transient: flushing, chest pain, dyspnea, bradycardia/asystole (brief), bronchospasm (avoid in severe asthma)
- AF triggered occasionally
- Wide-complex / pre-excited AF â avoid
Avoid
- Wide-complex SVT (use procainamide or cardioversion)
- WPW + AF (pre-excited AF)
- Severe asthma (bronchospasm)
- 2° or 3° AV block (worsens)
- Allergy
7ïžâ£ Catheter Ablation
Indications
- Recurrent symptomatic SVT
- WPW (especially symptomatic or high-risk)
- AF (Ch 264)
- VT in select cases (Ch 265)
- Atrial flutter
- Atrial tachycardia
- Idiopathic VT
Procedure
- Femoral venous access
- Catheters to right atrium â cross to left if needed (PFO or transseptal puncture)
- 3D mapping system (EnSite, CARTO)
- Radiofrequency or cryoablation
- 4-6 hours typically
- Outpatient or 1-day observation
Success Rates
- AVNRT: > 95%
- Typical AFL: > 95%
- AVRT (WPW): > 95%
- AF: 70-90% (paroxysmal); lower for persistent
- Idiopathic VT: 80-90%
- Scar-mediated VT: 60-80%
Complications
- Bleeding / vascular access
- AV block (especially AVNRT â slow pathway ablation; < 1% requiring pacemaker)
- Cardiac perforation / tamponade (rare)
- Atrial-esophageal fistula (AF ablation; rare but fatal)
- Stroke (rare)