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- è: Bordetella pertussis â gram - coccobacillus, fastidious, requires Bordet-Gengou or Regan-Lowe media, NAD-dependent
- Toxins: Pertussis toxin (PT) (AB5, ADP-ribosylate Gi), adenylate cyclase toxin (ACT), tracheal cytotoxin, FHA (filamentous hemagglutinin), LOS
- Reservoir: 人 only; highly contagious (R0 12-17, > measles)
- èšåº 3 phases (classic 6-12 wk):
- Catarrhal (1-2 wk): rhinitis, mild cough, low fever â most contagious phase
- Paroxysmal (2-6 wk): paroxysmal cough + whoop inspiratory + post-tussive vomiting, normal between bursts
- Convalescent (⥠2 wk): gradual decline; â100-day coughâ in adults
- 嬰å
< 6 mo: å€ atypical â apnea, cyanosis, no whoop; high mortality (pulmonary HTN, encephalopathy, secondary bacterial pneumonia)
- 2024 resurgence: USA, China, EU â 倧å¹
increase post-COVID immunity drop, waning vaccine immunity, anti-vaccine sentiment
- Treatment:
- Azithromycin 5d (preferred), Clarithromycin 7d, Erythromycin 14d
- TMP-SMX alt if macrolide allergy / R
- Best in catarrhal stage; paroxysmal stage abx äž alter clinical course but reduces transmission
- Prevention:
- DTaP infant series (2/4/6/15-18 mo + 4-6 yr booster)
- Tdap at 11-12 yr + once adult + æ¯æ¬¡ pregnancy 27-36 wk (passive infant immunity)
- æ¥è§žè
prophylaxis: azithromycin (same dose as treatment)
1ïžâ£ 现èåž
- Gram - coccobacillus
- Fastidious â needs Bordet-Gengou agar (potato-glycerol-blood) or Regan-Lowe (charcoal-blood)
- Slow grow (5-7 days)
- NAD-dependent
- Species:
- B. pertussis (#1 clinical)
- B. parapertussis â milder whooping cough
- B. bronchiseptica â kennel cough in dogs, rare human
Virulence
- Pertussis toxin (PT) â AB5 toxin, ADP-ribosylates Gi â cAMP â â impaired lymphocyte function + lymphocytosis
- Adenylate cyclase toxin (ACT) â entered host cells, â cAMP, impairs phagocytic + immune function
- Tracheal cytotoxin (TCT) â ciliated epithelium damage
- Filamentous hemagglutinin (FHA) â adhesion to ciliated cells
- Pertactin (PRN) â adhesin; some recent strains PRN-deficient â vaccine escape concern
- Lipo-oligosaccharide (LOS) â endotoxin
2ïžâ£ èšåºè¡šçŸ (Classic â Older Child / Adult)
Phase 1: Catarrhal (1-2 wk)
- Rhinitis, conjunctivitis, mild cough, low-grade fever
- Indistinguishable from URI
- Most contagious phase â but rarely diagnosed
- Lymphocyte å (PT effect)
Phase 2: Paroxysmal (2-6 wk)
- Paroxysmal cough: 5-15+ rapid consecutive coughs without inhalation between
- Inspiratory whoop (after paroxysm â air rushing through narrowed glottis)
- Post-tussive emesis
- Cyanosis, facial vein engorgement during paroxysm
- Normal interval between paroxysms (ç
人 looks well)
- ⥠14 days cough â classic case definition
Phase 3: Convalescent (⥠2 wk)
- Gradual decrease in paroxysm frequency + severity
- â100-day coughâ in adolescents/adults
- Triggered by URI later (waning effect)
Complications
- Pneumonia (primary or secondary bacterial â S. aureus, S. pneumo, H. influenzae)
- Rib fracture (from severe paroxysms)
- Urinary incontinence (paroxysm)
- Hernia
- Subconjunctival hemorrhage, epistaxis, syncope
- Pertussis encephalopathy (rare)
- Pertussis pneumonia in infants â high mortality
3ïžâ£ 嬰å
< 6 æ
- Atypical presentation â no whoop classic
- Apnea, cyanosis, bradycardia, gasping
- âSickâ infant with respiratory distress
- High mortality (1-3% in young infants)
- Secondary pulmonary hypertension + cardiopulmonary failure
- Encephalopathy
- Hospitalization standard
- Maternal Tdap during pregnancy is single most important protective measure for infant
4ïžâ£ æµè¡ç
åž
- Pre-vaccine: 200,000 cases/yr in US, 5000 deaths/yr
- DPT (1940s) â DTaP (1990s, less reactogenic but faster waning immunity)
- Waning immunity: 5-10 yr after vaccine; Tdap booster needed
- Resurgence in adolescents/adults â undetected reservoir â infant transmission
- 2024 surges:
- USA: 5-10Ã baseline cases
- China: 5000+ cases/wk early 2024
- EU: France, Spain, UK significant outbreaks
- Drivers: post-COVID immunity drop, waning DTaP, anti-vaccine, PRN-deficient strains (vaccine escape)
5ïžâ£ 蚺æ·
- PCR (nasopharyngeal swab) â most sensitive, especially first 3 wk
- Culture (Bordet-Gengou or Regan-Lowe) â gold standard but slow + low sens after 1st wk
- Serology â anti-PT IgG â late (⥠2 wk paroxysmal); not for early diagnosis
- Lymphocytosis (> 10,000-20,000) in classic pediatric â clue
- éå ± (notifiable)
6ïžâ£ æ²»ç
A. Antibiotic
- Best in catarrhal stage to alter clinical course
- After paroxysmal stage: still treat to reduce transmission (5 days post-abx no longer contagious)
- Macrolide first-line:
- Azithromycin 500 mg day 1 â 250 mg qd day 2-5 (10 mg/kg day 1 â 5 mg/kg day 2-5 for kids)
- Clarithromycin 500 mg bid à 7d
- Erythromycin 14d (worst tolerated, GI side effects)
- Alt: TMP-SMX 14d (macrolide allergy / R / azithro 倱æ)
- Newborn infant †1 month â erythromycin associated with pyloric stenosis; azithromycin å奜
B. Supportive
- Hospitalization for: < 6 mo, severe paroxysm, apnea, hypoxia, pneumonia, encephalopathy
- O2, suction, ICU monitoring
- IVF, nutrition
- Bronchodilators / steroids â no clear benefit (some try)
- Exchange transfusion + ECMO for severe pulmonary HTN in critical infants
7ïžâ£ Prevention
Vaccine Schedule (ACIP 2024)
- DTaP: 2 / 4 / 6 / 15-18 mo + 4-6 yr (5 doses) â pediatric
- Tdap: 11-12 yr + 1 in adulthood + æ¯æ¬¡ pregnancy 27-36 wk
- Td booster: q10y (Td or Tdap)
Pregnancy Tdap
- Each pregnancy 27-36 wk (regardless of prior)
- Maternal Ab â passive immunity to newborn
- ⥠80% reduction in infant pertussis < 6 mo
- Most important single intervention for infant protection
âCocooningâ Strategy
- All household contacts + healthcare workers + caregivers â Tdap up to date
- Reduces infant transmission