172.1 ð é«åžçç
172.1.0.1 ð äžé éé»
- è: Helicobacter pylori â Gram - curved/spiral rod, urease + (very strong), oxidase +, microaerophilic, motile (multiple polar flagella)
- Habitat: èé»è mucus å±€, urease å° urea â NH3 äžå stomach acid â æŽ»
- æµè¡ç åž: ~ 50%+ å šç; é«æåŸ declining, low-income still high
- Diseases:
- Asymptomatic colonization (倧å€)
- Chronic gastritis (universal in carriers)
- Peptic ulcer disease (PUD) â duodenal (90% link H. pylori) and gastric ulcer (80%)
- Gastric adenocarcinoma (Class I carcinogen per IARC)
- MALT lymphoma (eradication can cure early stage)
- ITP â eradication helps some
- Iron deficiency anemia â refractory cases
- Diagnosis:
- Non-invasive: urea breath test (UBT), stool antigen (high sens/spec); serology limited (not differentiate active vs past)
- Invasive (EGD biopsy): rapid urease test (CLO), histology, culture (for AST)
- Hold PPI ⥠2 wk before UBT/stool (PPI â urease activity â false neg)
- Treatment (2024 ACG / Maastricht VI):
- First-line: Bismuth quadruple (PPI + bismuth + tetracycline + metronidazole) Ã 14d â 90%+ cure
- OR Concomitant quadruple (PPI + amox + clari + metro à 14d) â è¥ clarithromycin R < 15%
- OR PCAB-based (vonoprazan + amox + clari, or P-CAB + amox dual)
- Clarithromycin triple äžå preferred (R é« globally)
- Test of cure: UBT or stool antigen ⥠4 wk after; off PPI ⥠2 wk
172.1.0.2 1ïžâ£ 现èåž + èŽç
- Spiral / curved gram - rod
- Urease極匷 â converts urea â NH3 + CO2 â buffer acid åšå â åæŽ» stomach
- Motile (flagella) â é² mucus å±€ (è¿ epithelium, pH ~ 7)
- Adheres to gastric epithelium via BabA (Lewis b binding)
- Virulence factors:
- CagA (cytotoxin-associated gene A) â type IV secretion â inflammation, carcinogenesis
- VacA (vacuolating cytotoxin) â induces vacuole + apoptosis
- OipA, BabA adhesins
- CagA + + VacA s1m1 = highest disease risk strain
172.1.0.2.1 Pathogenesis
- æ ¢æ§ inflammation â atrophic gastritis â intestinal metaplasia â dysplasia â adenocarcinoma
- Duodenal ulcer pathway: å€ antrum colonization â å¢ gastrin â å¢ acid â duodenal damage
- Gastric ulcer: å€ corpus colonization â atrophic gastritis â æž acid â epithelial damage
172.1.0.3 2ïžâ£ èšåºè¡šçŸ
172.1.0.3.2 B. Peptic Ulcer Disease
- Duodenal ulcer: ç epigastric, hunger/night pain, relief with food, H. pylori 90%+ when äž NSAID
- Gastric ulcer: ç worse with food, weight loss, H. pylori ~ 80%
- Complications: bleed, perforation, obstruction
172.1.0.3.3 C. Gastric Adenocarcinoma
- Class I carcinogen (IARC)
- Distal (non-cardia) adenocarcinoma mostly
- Long latency (decades)
- Endemic regions: Japan, Korea, China, Taiwan
- Eradication é incidence by ~ 50% in trials
172.1.0.3.4 D. MALT Lymphoma
- Gastric MALT (mucosa-associated lymphoid tissue) lymphoma
- ~ 90% H. pylori-driven
- Early-stage (stage IE) eradication â 70-80% complete regression
- Advanced + t(11;18) â eradication less effective; chemotherapy
172.1.0.4 3ïžâ£ 蚺æ·
172.1.0.4.1 Non-invasive Tests
| Test | Sensitivity | Specificity | Notes |
|---|---|---|---|
| Urea breath test (UBT) | 95%+ | 95%+ | First-line; hold PPI 2 wk |
| Stool antigen (monoclonal) | 95%+ | 95%+ | Equivalent to UBT; hold PPI 2 wk |
| Serology | 85-90% | 85% | Past vs active äžå; older test 挞 retire |
| Salivary | Variable | Research / public health |
172.1.0.4.2 Invasive (EGD Biopsy)
| Test | Notes |
|---|---|
| Rapid urease test (CLO test) | Quick, antrum + corpus biopsy; PPI false neg |
| Histology (Giemsa, IHC) | Gold standard; also see gastritis severity, dysplasia |
| Culture | Slow, but allows AST (susceptibility testing) for refractory cases |
| PCR | Detect organism + R mutations |
172.1.0.4.3 äœæ test (Indications â ACG 2017 + Maastricht VI 2022)
- Active or past PUD
- Gastric MALT lymphoma
- Early gastric CA after resection
- äžæ¥ dyspepsia < 60 yo + no alarm features â test-and-treat
- äžæ¥ GERD-only (not increased H. pylori risk)
- äžæ¥ NSAID users routinely (controversial; testing if high-risk for ulcer)
172.1.0.5 4ïžâ£ æ²»ç (2024 Updates)
172.1.0.5.2 A. Bismuth Quadruple à 14d (BQT)
- PPI bid + Bismuth subsalicylate 524 mg qid + Tetracycline 500 mg qid + Metronidazole 250-500 mg qid
- 90%+ cure rate
- Best for areas with clarithromycin R > 15% (most of globe)
- å¯äœçš: nausea, dark stool, metallic taste
172.1.0.5.3 B. Concomitant Quadruple à 14d (CQT)
- PPI + Amoxicillin 1 g bid + Clarithromycin 500 bid + Metronidazole 500 bid
- 90%+ cure if clari R < 15%
- 4 drugs daily for 14d â adherence issue
172.1.0.5.4 C. Vonoprazan-based (PCAB) Triple/Dual
- Vonoprazan 20 mg bid + Amox 1 g tid ± Clarithromycin 500 bid à 14d
- FDA 2022 (Voquezna Triple); PCAB more potent acid suppression
- Cure ~ 85% (triple), ~ 80% (dual without clari) in resistant strains
- No clari needed in dual â alternative for high R area
172.1.0.5.5 Not Preferred (Due Rising Resistance)
Clarithromycin triple (PPI + amox + clari à 7-14d)â cure 70% only when R > 15%Sequential 10d (PPI + amox 5d â PPI + clari + metro 5d)
172.1.0.5.6 Second-Line (Failed First-Line)
- If had clari â bismuth quadruple
- If had bismuth quadruple â levofloxacin triple (levo + amox + PPI Ã 10-14d)
- Rifabutin triple (rare; rifabutin + amox + PPI) â third-line