309.2 𩺠åèç
309.2.1 é«é »èé»
309.2.1.1 Inheritance + Mutations
- Autosomal recessive
- CFTR gene (chr 7)
- F508del: ~ 70% of alleles in Caucasian populations
- 6 functional classes (I-VI)
- Class I-III severe; IV-VI milder
309.2.1.2 Multi-Organ Manifestations
- Lung: bronchiectasis, infection, respiratory failure
- Pancreas: exocrine insufficiency (85-90%), CFRD (40-50% adult)
- GI: meconium ileus (newborn), DIOS (older)
- Liver: cirrhosis (10%)
- Sinus / nasal polyps
- Reproductive: CBAVD (male infertility 98%)
309.2.1.3 Diagnosis
- Sweat chloride ⥠60 mmol/L = positive
- 30-59 = borderline
- < 30 = negative
- Plus clinical features OR genetic testing
309.2.1.4 Lung Pathogens (Age-Stratified)
- Pediatric: S. aureus + H. influenzae
- Adolescent: Pseudomonas
- Adult: Pseudomonas + B. cepacia complex + NTM (MAC, M. abscessus)
309.2.1.5 CFTR Modulators (Revolutionary)
- Ivacaftor (Kalydeco, 2012): G551D class III
- Lumacaftor + ivacaftor (Orkambi, 2015): F508del homozygous
- Tezacaftor + ivacaftor (Symdeko, 2018): improved version
- ETI = elexacaftor + tezacaftor + ivacaftor (Trikafta/Kaftrio, 2019): F508del-containing genotypes; ~ 90% of CF patients eligible
309.2.1.6 CFTR Modulator Effects
- Improved lung function (FEV1 â 10-15%)
- Reduced exacerbations (~ 60%)
- Weight gain
- QOL improvement
- Reversed reproductive consequences (some)
309.2.2 ææ··æ·æ¯èŒ
| Feature | CF | Non-CF Bronchiectasis |
|---|---|---|
| Genetics | CFTR mutation | Variable (post-infectious, immune, etc.) |
| Sweat | Cl ⥠60 | Normal |
| Pancreas | Exocrine + endocrine insufficiency | Variable |
| Reproductive | CBAVD (male) | Variable |
| Sinus | Chronic + polyps | Variable |
| Pathogens | Pseudomonas, B. cepacia, NTM | H. influenzae, Pseudomonas (less) |
309.2.3 Special Topics
309.2.3.1 Pancreatic Sufficient vs Insufficient
- Pancreatic insufficient (PI): 85-90%; classical
- Pancreatic sufficient (PS): 10-15%; later onset, milder
- Determines PERT need
309.2.3.2 CFRD vs Type 1 vs Type 2 DM
- Combined insulin deficiency (β-cell destruction) + insulin resistance
- HbA1c misleading (faster RBC turnover)
- CGM essential
- Insulin only treatment (oral agents less effective)
- Microvascular complications (retinopathy, nephropathy)